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Considering the functional and structural similarities between the GCGR and GLP-1R, it was predicted that glucagon mediated these insulinotropic effects through signaling at the beta-cell GCGR

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identified impaired leukemia inhibitory factor/signal transduction (LIF/STAT3) pathway and disrupted endoplasmic reticulum stress in mice treated with excessive P4, leading to potential inhibition of embryo implantation and uterine transformation (Liang et al., 2018)

doi: 10.1152/ajpgi.00361.2020 164

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